(M80.3) Postsurgical malabsorption osteoporosis with pathological fracture

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1 643 865 in individuals diagnosis postsurgical malabsorption osteoporosis with pathological fracture confirmed
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39 170 deaths with diagnosis postsurgical malabsorption osteoporosis with pathological fracture
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2% mortality rate associated with the disease postsurgical malabsorption osteoporosis with pathological fracture

Diagnosis postsurgical malabsorption osteoporosis with pathological fracture is diagnosed Women are 68.17% more likely than Men

261 629

Men receive the diagnosis postsurgical malabsorption osteoporosis with pathological fracture

13 318 (5.1 %)

Died from this diagnosis.

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1 382 236

Women receive the diagnosis postsurgical malabsorption osteoporosis with pathological fracture

25 852 (1.9 %)

Died from this diagnosis.

Risk Group for the Disease postsurgical malabsorption osteoporosis with pathological fracture - Men and Women aged 80-84

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In Men diagnosis is most often set at age 0-19, 25-95+
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Less common in men the disease occurs at Age 20-24in in women, the disease manifests at any age
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In Women diagnosis is most often set at age 0-95+

Disease Features postsurgical malabsorption osteoporosis with pathological fracture

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Absence or low individual and public risk
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Postsurgical malabsorption osteoporosis with pathological fracture - what does this mean

Postsurgical malabsorption osteoporosis with pathological fracture occurs when a patient undergoes a surgical procedure that affects their ability to absorb nutrients from food, leading to a decrease in bone density and an increased risk of fractures. this can be further exacerbated by a lack of physical activity, a poor diet, and other medical conditions.

What happens during the disease - postsurgical malabsorption osteoporosis with pathological fracture

Postsurgical malabsorption osteoporosis with pathological fracture is the result of a combination of malabsorption of essential nutrients, such as calcium, and decreased bone density due to a surgical procedure. the malabsorption of essential nutrients can lead to decreased bone density, which can result in weakened bones and a higher risk of fracture. the fracture can be either spontaneous or due to a minor trauma. the fracture can also cause further damage to the bone, leading to further weakening of the bone and increasing the risk of fracture.

Clinical Pattern

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How does a doctor diagnose

  • Physical examination
  • Laboratory tests (e.g. complete blood count, serum electrolytes, serum calcium, phosphorus, alkaline phosphatase, vitamin D, vitamin B12, thyroid-stimulating hormone, parathyroid hormone, and 24-hour urinary calcium excretion)
  • Radiography (e.g. X-rays of the affected area)
  • Bone mineral density (BMD) test
  • Bone biopsy
  • Gastrointestinal function tests (e.g. fecal fat, vitamin B12 absorption)
  • Endoscopic evaluation (e.g. colonoscopy, upper endoscopy)
  • Small bowel imaging (e.g. CT scan, MRI)

Treatment and Medical Assistance

Main Goal: To reduce symptoms and prevent further bone loss and fractures.
  • Prescription of calcium and vitamin D supplements
  • Prescription of bisphosphonates to reduce bone resorption
  • Prescription of hormone replacement therapy
  • Prescription of bisphosphonates to reduce bone resorption
  • Prescription of antiresorptive medications
  • Prescription of anabolic agents to increase bone formation
  • Weight-bearing exercise program
  • Nutritional counseling
  • Physical therapy
  • Surgery to repair the fracture
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23 Days of Hospitalization Required
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Average Time for Outpatient Care Not Established

Postsurgical malabsorption osteoporosis with pathological fracture - Prevention

Prevention of postsurgical malabsorption osteoporosis with pathological fracture involves lifestyle modifications, adequate calcium and vitamin d intake, weight-bearing exercises, and medications such as bisphosphonates, calcitonin, and teriparatide. additionally, regular screening for bone density and fracture risk should be done to identify at-risk individuals and treat them accordingly.